What represents a chronic non-neurogenic cause of detrusor atony?

A chronic non-neurogenic cause of detrusor atony, such as functional urethral obstruction, leads to an inability of the detrusor muscle to contract effectively. In this scenario, the obstruction prevents normal bladder emptying over a prolonged period, which eventually results in overstretching and weakening of the bladder wall. The bladder may become distended and subsequently lose its ability to contract, ultimately leading to detrusor atony.

While psychogenic factors can influence micturition, they are not primarily responsible for causing the physical changes associated with detrusor atony. Acute bladder distension usually refers to a short-term condition, and while cystitis may lead to frequent urination, it typically does not cause detrusor atony on its own, as cystitis often involves irritation and inflammation rather than a chronic weakening of the bladder.

Therefore, the functional urethral obstruction stands out as a chronic cause that directly affects bladder mechanics, contributing to detrusor atony.